Respiratory complaints:
Asthma:
Usually known Hx of asthma.
Dyspnea with prolonged expiratory phase.
Expiratory wheeze.
Auscultation reveals wheezing or diminished/absent breath sounds.
Diminished SPO2
Elevated ETCO2
Usually a dry cough is present
Tripod positioning, accessory muscle use, or head bobbing.
Typically has an environmental trigger.
Treatments:
Salbutamol/albuterol (USA) MDI or Neb
Epinephrine 0.01mg/kg Max 0.5mg in patients with severe dyspnea requiring BVM and a known Hx of asthma ONLY if they are <50 years of age.
Supplemental oxygen.
COPD Exacerbation:
Usually there is a known Hx of COPD (emphysema, CB, or both).
Typically the patients are already on home O2 (Nasal cannula sign).
The patient usually has a chronic productive cough, and some level of dyspnea on exertion. However, during exacerbation this is going to be much more pronounced.
There will be a trigger for the exacerbation. Usually recent infections, fevers, allergen exposure, abnormal exercise level, or very hot or cold environmental exposures.
SPO2 will normally be high 80s to low 90s, but will likely be <88% in exacerbations.
ETCO2 of 50-60mmhg is not uncommon.
Treatments:
Supplemental O2 (increase above home O2 level if it is not effective).
CPAP in severe cases.
Salbutamol
12-lead ECG to rule-out cardiac causes of increased dyspnea.
Pneumonia:
Dyspnea that develops gradually over several hours to days.
Sharp, diffuse, pleuritic chest pain.
Cough, typically productive of yellow, green, or while sputum.
Rhonchi on auscultation.
Fever or chills (currently or recently).
Often occurs in the setting of other comorbidity such as COPD or asthma, or in immunocompromised patients, or those with respiratory difficulties (post-surgery, neuromuscular disorders, etc).
Treatments:
Supplemental oxygen.
Salbutamol
IV cannulation and potential bolus if the patient is hypotensive (sepsis).
Passive temperature reduction in the setting of fever.
Pulmonary Embolism:
Sudden sharp, well localized (“pinpoint”) pleuritic chest pain.
Dyspnea, with clear lung sounds throughout.
Low SPO2
LOW ETCO2
Hypotension
Tachycardia
DVT or recent history suggestive of one.
Treatments:
Supplemental O2.
12-lead ECG to rule out MI and look for changes that support PE (Right heart strain pattern).
IV cannulation and potential bolus.
Rapid transport.
Pneumothorax:
Sudden onset of sharp pleuritic chest pain on the affected side.
Unilateral breath sounds (diminished or absent over affected region).
Low SPO2.
Dyspnea
Tachycardia
Hypotension if Tension Pneumothorax develops (especially with mediastinal shift).
Possible subcutaneous emphysema.
Tracheal deviation (late sign).
ALS may provide needle decompression.
Treatments:
Supplemental O2, and potentially PPV with a reduced tidal volume.
IV cannulation in anticipation of potential hypotension.
Rapid transport.
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